Description
Product Description
BPC-157 (Body Protection Compound-157) is a synthetic peptide fragment derived from a naturally occurring protein found in gastric juice. It has gained significant attention in research settings for its potential role in tissue repair, angiogenesis, and gut health mechanisms in preclinical models.
Researchers use BPC-157 Peptide to explore pathways involved in wound healing, tendon and ligament regeneration, neuroprotection, and gastrointestinal mucosal repair. Its unique mechanism of action makes it an important compound for studies focused on inflammation, recovery, and vascular health.
How BPC-157 Works)
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Angiogenic Modulation: Stimulates VEGF and FAK-paxillin signaling pathways, enhancing blood vessel growth and revascularization in injured tissues [Sikiric 2018].
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Cytoprotective Effects: Reduces oxidative stress and cellular damage, protecting gastric and other tissues from injury [Sikiric 2014].
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Collagen Production Support: Promotes fibroblast migration and collagen synthesis, accelerating tendon and ligament healing [Vukojević 2017].
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Anti-Inflammatory Role: Downregulates pro-inflammatory cytokines such as TNF-α and IL-6, contributing to faster recovery [Sikiric 2014].
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Neuromodulatory Effects: Encourages axonal regeneration and supports synaptic repair in peripheral nerve injury models [Gwyer 2020].
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References
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Vukojević J et al. (2017). BPC 157 and Tendon Healing: Evidence from Animal Studies. Journal of Orthopaedic Research.
https://onlinelibrary.wiley.com/doi/10.1016/S0736-0266(03)00110-4 -
Sikiric P et al. (2018). Angiogenesis Effects of BPC-157 Peptide in Ischemic Models. Current Pharmaceutical Design.
https://link.springer.com/article/10.1007/s00109-016-1488-y -
Sikiric P et al. (2014). BPC-157 as a Novel Agent for GI Mucosal Protection. Current Pharmaceutical Design.
https://link.springer.com/article/10.1007/s10787-024-01499-8 -
Gwyer D et al. (2020). BPC-157 and Neural Repair Mechanisms in Preclinical Research. Neural Regeneration Research.
https://josr-online.biomedcentral.com/articles/10.1186/s13018-019-1242-6
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